Pig heart is sweet and salty; nature is flat;
effect
Yang Xin soothe the nerves, benefit the spleen and stop the bleeding, set the startle, cure convulsions, phlegm, spontaneous sweating, insomnia.
Taboo crowd
Those with hypercholesterolemia should not eat;
people suitble
suitable for majority of the people. Especially suitable for guilty perspiration, spontaneous sweating, convulsions, jealousy, insomnia and dreams; schizophrenia, epilepsy, and rickets.
Pig heart is the heart of the pig and is a tonic food. Is a very rich food nutrition.
It contains protein, fat, calcium, phosphorus, iron, vitamin B1, vitamin B2, vitamin c, and nicotinic acid, which have a great effect on strengthening the heart muscle nutrition and enhancing the myocardial contractility. Commonly used for abnormalities of the heart. With the application of drugs to relieve heartburn, the effect is obvious.
The function and effect of pig heart
Effects on the cardiovascular system
Atrial natriuretic peptide (Anp) can be extracted from pig atrium, which can selectively relax blood vessels and lower blood pressure. The renal artery and isolated aorta are most sensitive to ANP, while the mesenteric artery, coronary artery, femoral artery, pusher artery, and carotid artery are less sensitive or insensitive. Anti-arrhythmia, increased myocardial contractility, increased cardiac output, expansion of coronary and peripheral blood vessels, lower blood pressure and other effects.
The role of the kidney
Atrial peptides have strong sales and diuretic effects. At the same time, urinary excretion of Cl-, Ca2+, Mg2+, K+, and phosphates increases. K+ and Ca2+ discharges are relatively low, and HCO-3 does not change, generally not affecting blood concentration. The mechanism of ANP diuretic sales may be:
(1) Increased renal blood flow and glomerular filtration rate. It has been reported that ANP can reduce the resistance of the afferent arterioles, increase the resistance of the arterioles, increase the glomerular filtration rate and the filtration rate of a single nephron. .
(2) Suppression of reabsorption of Na+ by renal tubules and collecting ducts. It is also believed that ANP can still enhance the secretory effect of renal tubules.
(3) Inhibition of the renin-angiotensin-aldosterone system, ANP inhibits the secretion of renin and aldosterone.
(4) Inhibition of synthesis and release of antidiuretic hormone.
(5) ANP may bind to specific receptors on the cell surface, activate bird cyclase, increase cytosolic CGMP content, cGMP is the second messenger of ANP diuretic sodium. Intravenous injection of ANP to patients with pregnancy induced hypertension caused a significant drop in blood pressure, but also significantly reduced or disappeared edema and urine protein.
Effects on energy metabolism and substance metabolism
Pig heart can be used to extract cytochrome C, which can enhance cell respiration. Exogenous C does not pass through normal cell membrane, but cell membrane permeability increases in hypoxia and can enter cells, directly participate in mitochondrial respiratory chain, and play an important role in electron transfer. The role of the body, so that cell respiration, enhance the utilization of oxygen, enhance tissue metabolism.
C can improve cell metabolism in aged rats. Both intramuscular injection and oral administration can make the decreased oxidative phosphorylation index (P/O) rise. C can also improve lipid metabolism and glucose metabolism in aged rats. Oral or intramuscular injection can significantly reduce the serum lipids and hepatic lipids in aged rats, especially the total lipid and cholesterol levels in the liver. Improve the ability of oxidative fatty acids in liver mitochondria in aged rats. Aged rats lack glucose metabolism, blood glucose, liver glycogen and serum niacin concentrations are significantly reduced, given C can improve its glucose metabolism.
Anti-oxidize effect
CoQ10 in the heart of pig is an important natural antioxidant that can effectively inhibit the rapid hemolysis of rabbit red blood cells induced by vitamin A alcohol and enhance the anti-hemolytic effect of red blood cells in preterm infants. COQ10 has a free radical scavenging effect. Free radicals are particles containing an unpaired electron. The electrons have a strong ability to oxidize and attack the molecules they encounter. There are many ways to produce free radicals in the body during ischemia, causing changes in cell membrane function and structure, resulting in cell damage and necrosis.
In rats and large experiments, it was confirmed that COQ10 scavenging free radicals can significantly reduce myocardial mitochondrial oxygen consumption, reduce cellular ATP consumption, maintain cell mitochondrial structural integrity, prevent cell edema and cell membrane rupture, and mitochondrial lysis and myogenic The arrangement of fibers is disordered.
Detoxification
Stabilizing hepatocyte microsomal membranes reduces the amount of tyrosine that enters the microsomes and reduces the production of toxic metabolites, thereby reducing liver cell damage. The pig heart can also resist the liver damage caused by CCl4 in rats, making serum lipid peroxides and ALT significantly lower than the control group, and better than Gantai Yue, its lesion degree is also lighter than the control group. Oral Cyt-C vs. CCL. The liver damage caused by the rats also has a significant antagonistic effect, so that hepatocyte vacuolation and necrosis, cell infiltration, increased fat droplets, decreased glycogen particles, rough endoplasmic reticulum degeneration and reduction of other diseases have significantly reduced. The cardiotoxicity of porcine heart on adriamycin also has a significant antagonistic effect, which makes the changes of electrocardiogram, blood urea nitrogen and creatine induced by doxorubicin return to normal, and can prevent myocardial cells caused by adriamycin AT. .
Anti-tumor effect
CoQ10 in pig hearts is used to prevent the incidence of inoculated tumors. It can be used for treatment to delay the growth and metastasis of tumors and prolong survival. Injecting CoQ10 into mice significantly increased the resistance to diphenylhydrazine-induced tumor and leukemia caused by Friend leukemia virus infection, resulting in a decrease in the number of mice with cancer, increased survival, and reduced tumor size. For rat AH13 liver cancer, rat NoVikoff liver cancer and mouse Friend leukemia, CoQ10 can significantly inhibit the oxygen consumption of malignant tumor cells in vitro, and can correct CoQ10 in cancer cells in vivo. Lack of state reduces the host's condition and reduces mortality. In patients with advanced metastatic cancer, COQ10 can stimulate its immune system, stabilize the disease and prolong survival.
Relaxation of intestinal smooth muscle
Capillary vascular permeability increases, blood volume decreases, erythrocyte permeability, blood viscosity, and plasma colloid osmotic pressure increase. ANP on oleic acid-induced pulmonary edema in rats and respiratory distress syndrome in rats can significantly relieve the symptoms, can reduce pulmonary interstitial edema and atelectasis, wells to increase the concentration of surfactant in the lung.
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